The scientific productivity of the B3OA laboratory includes research and clinical publications and is characterized by (i) an increase in the total number of publications from 236 in 2007-2012 to 371 in 2012-2017 and (ii) an increase in the number of publications considered exceptional (from 37 in 2007-2012 to 44 in 2012-2017) and excellent (from 57 in 2007-2012 to 112 in 2007-2017) by INIST CNRS.
Our latest publications
Concise review: Understanding and leveraging cell metabolism to enhance mesenchymal stem cell transplantation survival in tissue engineering and regenerative medicine applications.
Here, we provide a comprehensive review of plausible metabolic switches in response to implantation and of the various strategies currently used to leverage MSC metabolism to improve stem cell-based therapeutics.
Effect of the bone morphogenetic protein-2 doses on the osteogenic potential of human multipotent stromal cells-containing tissue engineered constructs
Our results showed that the addition of only low dose of BMP-2 was beneficial to enhance the bone-forming potential of MSCs, whereas high dose of BMP-2 overcame the advantage of combining this growth factor with MSCs.
In this uncontrolled study, airway bioengineering using stented aortic matrices demonstrated feasibility for complex tracheal and bronchial reconstruction.
Method and device for ultrasound testing of the mechanical strength of a part inserted in a body, in particular of a dental implant
The invention relates to a method for controlling the mechanical strength of a part inserted at least partially in a body.
The invention relates to an implant device and a delivery tool.
The invention relates to a method for culturing bone tissue.
The present invention relates generally to a hydrogel releasing glucose in a time-controlled manner, to medical applications thereof, and to a method for preparing said hydrogel.
Type 2 diabetes impairs angiogenesis and osteogenesis in calvarial defects: MicroCT study in ZDF rats
The present study provided evidence that T2DM impairs bone formation in critical-size calvarial defects and markedly reduces angiogenesis in all defects regardless of the defect size tested.